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During wound healing, MMPs play dual roles: they facilitate tissue debridement and cell migration in early healing phases, while excessive or prolonged MMP activity can impair matrix deposition and delay closure

CHE showed potent anticancer activity against TNBC that overexpressed PKN2 and PRKCA by inhibiting cell growth and inducing apoptosis (Lin et al., 2017), selectively inhibiting growth with a half-inhibitory concentration (IC 50 ) of 1.6 M for MDA-MB-231 cells and increasing ROS generation, thereby inducing apoptosis (Matkar et al., 2008

The long-term safety issues associated with the chronic administration of oral chelators require consideration